Dementia is considered as a condition where a person suffers from a mere decline in mental capacity to a serious enough loss, that can reduce quality of life. Memory loss is one example.
Dementia does not concern specific diseases. It is a set of symptoms related to memory loss or other thinking skills severe enough to reduce a person’s ability to perform daily activities. Alzheimer’s disease accounts for 60-80% of cases. Vascular dementia, which occurs after a stroke, is the second most common type of dementia. But there are many other conditions that can cause dementia, including some that are reversible, such as thyroid problems and vitamin deficiencies. The various forms of dementia are often generally and clearly misidentified as “old” or “senile dementia”, which reflects the former widespread but erroneous belief that severe spiritual decline is a normal part of aging.
Diagnosis of Real Causes & Treatments of Dementia
- Gradual restoration of cellular function
- Personalized therapeutic protocols, without chemical residues and excipients
- Treating the real causes
- Therapeutic formulas that work alone or in combination with any other medication
- Adopting a Molecular / Therapeutic Nutrition Plan
The epidemiology of dementia does indeed reveal that these diseases have reached epidemic proportions and cause a serious public health problem. They occur in about 6% of people over 65 years of age and are highly age-dependent. In the 65-69 age group, there are more than two new cases per 1000 people each year. This number is increasing almost exponentially with increasing age from the age of 90, out of 1000 people, 70 new cases of dementia are expected annually.
Memory loss and other dementia symptoms
While symptoms of dementia can vary widely, at least two of the following basic spiritual functions must be significantly attenuated in order for individuals to be considered ill. These can be: memory, communication and language, focus and attention, critical ability, visual perception.
Causes of dementia
Dementia is caused by damage to the brain cells. These lesions impede the ability of brain cells to communicate with one another. When brain cells cannot communicate properly, thinking, behavior, and emotions can be affected. The main difference between age-related memory loss and dementia is that memory loss has little effect on daily performance and abilities, while dementia, on the other hand, is characterized by a persistent, neurodegenerative process affecting two or more mental faculties such as memory, language, judgment and abstract thinking. Some causes of dementia-like dementia or symptoms can be reversed with treatment. These include:
- Infections and disorders of the immune system.
- Metabolic problems and endocrine abnormalities.
- Eating deficiencies.
- Side effects of medicines.
- Subcutaneous hematomas.
- Heavy metal poisoning, such as lead
Types of dementia
Different types of dementia relate to specific types of brain cell damage in specific areas of the brain. For example, in Alzheimer’s disease, high levels of certain proteins inside and outside the brain cells make it difficult for the brain cells to stay healthy and communicate with one another. The brain region called the hippocampus is the center of learning and memory in the brain, and the brain cells in that region are often the first to be damaged. That is why memory loss is often one of the first symptoms of Alzheimer’s disease.
While most dementia-induced brain changes are permanent and worsen over time, thought and memory problems caused by depression, drug side effects, alcohol overuse, thyroid problems, vitamin deficiency can be improved when the exact cause is addressed or identified.
How is it treated so far?
The drugs used to date help the brain’s memory system improve its functioning. They consist of 2 categories:
- acetylcholinesterase inhibitors (donepezil, ribastigmine, galantamine)
- NMDA receptor inhibitors (the memantine)
These substances improve mental functioning in 2/3 of patients up to 12 months. They also improve the psychiatric symptoms and behavioral disorders as well as the functionality of the individual. Medications that improve other symptoms (eg, antidepressants if depressed, depressant in anxiety) or are used to treat, for example, correction of abnormalities in sodium, calcium, thyroxine administration, hypothyroidism or neovascularization, subdural hematoma.
Genetic predisposition to dementia
Some risk factors, such as age and genetics, cannot be changed. However, researchers continue to investigate the effect of other risk factors on brain health and prevention of dementia. Some of the most active research areas for risk reduction and prevention include cardiovascular factors, fitness and nutrition.
It is not just a gene, but the interaction between many genes and the environment that puts one at risk for chronic diseases, such as dementia. Also we know that many factors affect the function of genes such as nutrition, vitamins and minerals, toxins, allergens, stress, lack of sleep and exercise and much more. A complete gland mapping is capable of identifying the responsible genes for the disease. Early diagnosis can cure reversible causes of memory loss, reduce dementia or improve the quality of life of people with Alzheimer’s disease or other forms of Alzheimer’s disease.
The new medical reality
Restoring proper thyroid function, treating mercury toxicity, treating chronic inflammation and deficiencies in vitamins B6 and D, folic acid, coenzyme Q10 and omega-3 fatty acids, natural hormone or natural hormones guidance greatly helps improve cognitive function and prevent dementia. Dementia in modern medical reality can be reversed by monitoring all the factors that affect the function of the brain, with proper genetic compliance with all the conditions of the body to work in a balanced and nutrient-rich manner.
Dr. Nikoleta Koini, M.D.
Doctor of Functional, Preventive, Anti-ageing and Restorative Medicine.
Diplomate and Board Certified in Anti-aging, Preventive, Functional and Regenerative Medicine from A4M (American Academy in Antiaging Medicine).
- Stroombergen, M.C., and R.H. Warring. (1999). Determination of glutathione S-transferase me and theta polymorphisms in neurological disease. Human and Experimental Toxicology. 18 (3):141-145.
- Sierpina VS1, Sierpina M, Loera JA, Grumbles L. South Med J. 2005 Jun;98(6):636-45. Complementary and integrative approaches to dementia.
- Kat Toups Title: Dementia Demystified: A Functional Medicine Approach to Reversing Cognitive Decline and Dementia. Bay Area Wellness – Functional Medicine Psychiatry and Dementia, USA Aging (Albany NY). 2014 Sep;6(9):707-17.
- Matthews, K. A., Xu, W., Gaglioti, A. H., Holt, J. B., Croft, J. B., Mack, D., & McGuire, L. C. (2018). Racial and ethnic estimates of Alzheimer’s disease and related dementias in the United States (2015–2060) in adults aged≥ 65 years. Alzheimer’s & Dementia. https://doi.org/10.1016/j.jalz.2018.06.3063External
- Heron M. Deaths: leading causes for 2010. National vital statistics reports; vol. 62, no 6. Hyattsville, MD: National Center for Health Statistics. 2013.
- James BD. Leurgans SE, Hebert LE, et al. Contribution of Alzheimer disease to mortality in the United States. Neurology. 2014;82:1-6.
- Nelson AR, Sweeney MD, Sagare AP, Zlokovic BV. Neurovascular dysfunction and neurodegeneration in dementia and Alzheimer’s disease. Biochim Biophys Acta. 2016 May;1862(5):887-900. doi: 10.1016/j.bbadis.2015.12.016.
- Giannopoulos PF, Praticò, D. Alzheimer’s Disease. In: Martin CR, Preedy VR eds., Diet and Nutrition in Dementia and Cognitive Decline. London: Elsevier; 2015: p.38-46.
- Bredesen, D. (2017, December). Clinical Case Studies with Subtypes. Presented at the Institute for Functional Medicine Reversing Cognitive Decline Advanced Clinical Training, Miami, Florida.